Docosahexaenoic Acid (DHA) is what we call an ortho nutrient, meaning it is an essential component of proper functioning of the human body. Every cell membrane in the body contains DHA as part of its chemical composition and accumulates intensely in the brain & retina. Over 50% of a neuron’s plasma membrane is DHA, and ~15% of the total fatty acid composition of the pre-frontal cortex is DHA. Common dietary sources of DHA include various fish, breast milk, and algae.
Vegetarians and vegans can have an especially difficult time getting enough of this essential omega-3, as most plant-based omegas are in the form of alpha-linolenic acid (ALA). ALA undergoes limited conversion in the liver to DHA/EPA; however, genetic predisposition and aging both greatly limit the conversion amount. Pregnant or nursing mothers are another high-risk group for DHA deficiency, as the mother passes enormous amounts of DHA to the child to encourage proper brain & retinal development. Personally, I prefer taking an algal-based DHA oil, as it is confirmed free of PCB’s & heavy metals, and does not harm fish to create. Algal DHA has been shown to have the same bioavailability as its fishy counterpart. Symptoms of omega-3 deficiency include learning disabilities, depression, arthritis, eczema, obesity, & heart disease.
A huge problem in our society is the mass consumption of unhealthy trans-fatty acids. Trans-fats are liquid vegetable oils that have undergone hydrogenation, causing the oil to become a solid at room temperature. Direct links exist between the consumption of trans-fats and Alzheimer’s, cancer, diabetes, obesity, liver dysfunction, infertility, depression, irritability, and aggression. These toxic fats get stuck in the cell’s membrane and disrupt its normal functions. Toxins that don’t belong make their way into the cell, and nutrients begin to leak out. Compromising the membranes of one’s cells hardly seems like a solid plan for longevity. Major culprits for these trans-fats include potato chips, fast food, fried food, margarine, shortening, cake mixes, frozen foods, donuts, & microwaveable popcorn. Though, if you are far enough along on your quest to be reading this information, you probably don’t eat any of the foods mentioned above. Good on you!
So, if you find yourself in a cellularly compromised situation, grab some shilajit & a capsule of DHA. The Pürblack Live Resin offers incredibly potent, deeply detoxifying effects, as well as helping in the assimilation of nutrients. It could [theoretically] help to usher poisonous trans-fats out of your precious membranes, and welcome in the life-giving DHA. This regimen of Pürblack & DHA is so broad in its medicinal scope that most anyone would probably see vast improvements in all markers of health & well-being. They work in tandem to affect virtually every single aspect of human physiology. Various studies have shown that DHA upregulates cortical neurogenesis (1), expression of neurotrophic factors (2), as well as nerve growth factor (NGF) induced neurite regeneration & synaptogenesis (3).
When choosing a DHA supplement, look for one that has a 3:1 ratio of DHA:EPA. The average human has a very low need for EPA relative to DHA, yet ironically, most supplements contain the opposite ratio. Estimates state that as much as 90% of Americans are omega-3 deficient! After supplementing with DHA for 2-3 weeks, I began to notice enhanced color saturation, mental clarity, drastically reduced anxiety, and a more pronounced feeling of being in the present moment. Don’t be another statistic, embrace Pürblack & DHA as part of your repertoire for optimum vitality and vigor!
1. Coti Bertrand P, O’Kusky JR, Innis SM. Maternal dietary (n-3) fatty acid deficiency alters neurogenesis in the embryonic rat brain. J Nutr. 2006;136:1570–5.
2. Ikemoto A, Nitta A, Furukawa S, Ohishi M, Nakamura A, Fujii Y, Okuyama H. Dietary n-3 fatty acid deficiency decreases nerve growth factor content in rat hippocampus. Neurosci Lett. 2000;285:99–102.
3. Martin RE, Bazan NG. Changing fatty acid content of growth cone lipids prior to synaptogenesis. J Neurochem. 1992;59:318–25.